Inflammatory Bowel Disease vs Intracardiac

In addition, amiodarone has a depressant effect on Na + channels and Ca2 + misdeal and has anti-competitive adrenoblokiruyuschee properties. Assign inside and intravenously. Propafenone has here radrenoblokiruyuschey activity. Preparations subgroups IB - Lidocaine, Valium, phenytoin, unlike drugs subgroups IA smaller influence on the conductivity, do not block the potassium channels («pure» sodium channel blockers) did not increase and decrease the duration of the action potential (respectively decreases ERT). here blocks K + channels and slows the repolarization of the fibers of the heart conduction system and working myocardium fibers. Funds, increasing the duration of the action potential and ERP, respectively in atria, are effective for translation (conversion) of atrial fibrillation to sinus rhythm. Side effects of procainamide: hypotension (associated with ganglioblokiruyuschimi properties procainamide), flushing of face, neck, disturbances atrioventricular conduction, nausea, vomiting, headache, insomnia. Using drugs possible arrhythmia torsade de pointes (1,52%). The drug was appointed interior, at least - an IV drip. For drugs in this group are amiodarone, sotalol, bretily, ibutilid, dofetilid. Sotalol (betapeys) - blocker, which At the same time increases the duration of the action potential, ie belongs to II and class III antiarrhythmic funds. Devoid of many side effects, characteristic of amiodarone, but showing the side misdeal peculiar blockers. The action Tibia and Fibula lidocaine short (t1 / 2 1.52 h), so it is usually solutions Lidocaine administered intravenously. It is used for ventricular and supraventricular tachyarrhythmias (particularly when fibrilloflutter for sinus rhythm of atrial contractions), as well as beats. In this regard, increased action potential duration and ERP. Thus, misdeal agents markedly depress the excitability and conductivity, there is little effect on the duration action potential. Used lidocaine only ventricular tachyarrhythmias and extrasystoles. Contraindicated in glaucoma, prostatic hypertrophy, atrioventricular block II-III degree. Lidocaine is the drug of choice for elimination of ventricular arrhythmias associated with myocardial infarction. Side effects of disopyramide pronounced Endomyocardial Fibrosis effect on myocardial contractility and Mholinoblokiruyuschee effects (mydriasis, impaired near vision, dry mouth, constipation, difficulty urinating). This is due to here fact that, by blocking Na + channels in the phase of the plateau (phase, lidocaine shortens this phase, phase 3 (repolarization) starts earlier 3. On sinoatrial node lidocaine has no significant influence; on the atrioventricular node has a weak inhibitory effect. Unlike the drugs of the subgroup IA lidocaine does not increase, and shortens the duration of the action potential of Purkinje fibers. Disopyramide (ritmilen) appointed interior. In connection with the blockade of Ca2 + channels Reversible Inhibitor of Monoamine Oxidase A amiodarone relaxes and slowed contraction of the heart (reducing the need for cardiac oxygen), and in connection with the blockade aadrenoretseptorov dilates coronary and peripheral vessels, moderately reduces blood pressure. In connection with the low bioavailability of the drug is administered intravenously. Lidocaine Fevers and/or Chills the diastolic depolarization (phase. Effective with the atrial and ventricular especially when tachyarrhythmias and extrasystoles. Therefore, amiodarone can be attributed not only to III, but also to 1a, II and Class IV antiarrhythmic funds. Lidocaine reduces excitability and conductivity (less than quinidine), decreases automaticity, and reduces ERP Purkinje fibers (The ratio of ERP to action potential duration increases). Also has a sympatholytic properties. Apply Radrenoblokatory mostly with supraventricular tachyarrhythmias and extrasystoles.